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max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Conversely, our power to rescue SERT operate in KI midbrain synaptoneurosomes by the inhibition of FAK signifies elevated FAK signaling downstream from the Pro32Pro33 mutant, as verified by elevated pFAK localization in five-HT synapses. Our i